Best Postdoctoral Publication Awards
The Postdoctoral Assembly (PDA) of the Society of Toxicology is pleased to announce the Best Postdoctoral Publication Awards (BPPA). These awards were created to recognize talented postdoctoral researchers who have recently published exceptional papers in the field of toxicology. Applications are reviewed by the PDA Board and a committee containing appropriate scientific expertise. The review process follows National Institutes of Health (NIH) conflict of interest policy, confidentiality, and nondisclosure policies. Three awards, each consisting of a plaque and a $100 cash award, are presented at the PDA Luncheon during the SOT Annual Meeting.
Previous BPPA Award Recipients
Eligibility for 2011 Nominations
- The research reported in the paper was conducted while the applicant was engaged in a postdoctoral research experience
- Member of SOT or membership application submitted by award application deadline
- First author on a peer-reviewed paper published on-line or in print, or in press, between June 1, 2009, and May 31, 2010
- Application supported by letter from the research advisor
- Only one paper may be submitted by each applicant
- Leaders of the PDA are eligible but applicants will not participate in the award determination
Applicants
Items should be assembled in this order as one PDF file and submitted by the applicant.
- A completed Best Postdoctoral Publication Award Application Form
- A statement explaining the significance of the result(s) and indicate how these results impact the advancement of science (length no more than one page)
- Provide a copy of the nominated paper
- Letter of acceptance from the editor if the paper is in press
Advisor Nomination
Items should be in this order as one PDF file submitted by the advisor.
- A completed Best Postdoctoral Publication Advisor Nomination Form to certify the contributions of the applicant
- A statement describing the significance of the work and detail concerning the contributions of the nominee to the research in the paper (length no more than one page)
Application materials must be sent via e-mail to SOT Headquarters before 12:00 MIDNIGHT EST October 9, 2010.
Award Recipients
2010 Recipients
Bret F. Bessac, Yale University School of Medicine
Transient Receptor Potential Ankyrin 1 Antagonists Block the Noxious Effects of Toxic Industrial Isocyanates and Tear Gases
The FASEB Journal (2009) Vol. 23 1102-1114
Manabu Nukaya, University of Wisconsin-Madison
The Role of the Dioxin Responsive Elements Cluster Between Cyp1a1 and Cyp1a2 Loci in Aryl Hydrocarbon Receptor Biology
Proc. Natl. Acad. Sci. USA (2009) 106, 4923-8
Nicholas M. Radio, Cellumen
Assessment of Chemical Effects on Neurite Outgrowth in PC12 Cells Using High Content Screening
Toxicol Sci. (2008) 105(1),106-118
2009 Recipients
Jeffrey Card, Cantox Health Services International
Cyclooxygenase-2 Deficiency Exacerbates Bleomycin Induced Lung Dysfunction but not Fibrosis
Am J Respir Cell Mol Biol 2007 Sep, 37(3):300-8
Kembra Howdeshell, NHEERL, USEPA
A Mixture of Five Phthalate Esters Inhibits Fetal Testicular Testosterone Production in the Sprague-Dawley Rat in a Cumulative, Dose-Additive Manner
Toxicol Sci. 2008, 105:153-165
Lewis Shi, University of Wisconsin-Madison
The Aryl Hydrocarbon Receptor Is Required for Optimal Resistance to Listeria monocytogenes Infection in Mice1
The Journal of Immunology 2007, 179: 6952-6962
2008 Recipients
Joshua Gray, University of Medicine and Dentistry of New Jersey
Paraquat Increases Cyanide-insensitive Respiration in Murine Lung Epithelial Cells by Activating an NAD(P)H:Paraquat Oxidoreductase: Identification of the Enzyme as Thioredoxin Reductase.
Journal of Biological Chemistry Vol. 282, NO. 11, pp. 7939-7949, March 16, 2007.
Christie Sayes, DuPont Haskell Laboratory for Health and Environment
Assessing Toxicity of Fine and Nanoparticles: Comparing In Vitro Measurements to In Vivo Pulmonary Toxicity Profiles.
Toxicol Sci. 97(1), 163–180 (2007).
Khristy Thompson, Elizabethtown College
Olfactory Uptake of Manganese Requires DMT1 and is Enhanced by Anemia.
The FASEB Journal (January 2007) Vol. 21, 223–230.
2007 Recipients
Nadine Dragin, University of Cincinnati
Dioxin-Induced Birth Defects, Mouse or Human CYP1A2 in Maternal Liver Protects whereas Mouse CYP1A1 and CYP1B1 are Inconsequential.
The Journal of Biological Chemistry, VOL. 281, NO. 27, pp. 18591-18600, July 7,
2006.
Kristen Mitchell, University of Texas Medical Branch
Sustained Aryl Hydrocarbon Receptor Activity attenuates Liver Regeneration.
Mol Pharmacol. 2006 Jul;70(1):163-70. Epub 2006 April 24, 2006.
Drobna Zuzana, University of North Carolina at Chapel Hill
Metabolism and Toxicity of Arsenic in Human Urothelial Cells Expressing Rat Arsenic (+3 oxidation state)-methyltransferase.
Toxicology and Applied Pharmacology 207 (2005) pp. 147-159, 2005.
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